30-31st of Oct 2023
Purpose of the meeting: To bring together for the first time the members of the COPDiNET network for scientific exchange and to promote collaborations in the field.
Scope: The meeting will cover latest research and clinical advances in COPD and chronic lung disease research and technological advances that will facilitate research in these areas
Location: Schloss Hohenkammer (Bavaria /Germany) https://www.schlosshohenkammer.de/
PRELIMINARY AGENDA:
Talks format:
- Invited speakers 15min talk +5min discussion.
- Selected from abstract: 7min talk + 3min discussion.
Organising committee:
Ali Önder Yildirim, Roxana Wasnick, Thomas Conlon, Reinoud Gosens, Theodoros Kapellos, Mareike Lehmann, Enid Neptune, Francesca Polverino, Maor Sauler
Yildirim/Conlon Lab, LHI
Immunopathology of COPD
We want to understand how the immune system drives tissue injury and modulates tissue repair and regeneration in COPD to devise novel therapeutic targets to ultimately reverse chronic lung disease – particularly COPD.
Slide 2
Understanding COPD pathogenesis and therapeutics using cellular, mouse, and human models by tobacco and other environmental exposures
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Understanding COPD pathogenesis and therapeutics using cellular, mouse, and human models by tobacco and other environmental exposures
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Understanding COPD pathogenesis and therapeutics using cellular, mouse, and human models by tobacco and other environmental exposures
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Understanding COPD pathogenesis and therapeutics using cellular, mouse, and human models by tobacco and other environmental exposures
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Understanding COPD pathogenesis and therapeutics using cellular, mouse, and human models by tobacco and other environmental exposures
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Understanding COPD pathogenesis and therapeutics using cellular, mouse, and human models by tobacco and other environmental exposures
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Understanding COPD pathogenesis and therapeutics using cellular, mouse, and human models by tobacco and other environmental exposures
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Understanding COPD pathogenesis and therapeutics using cellular, mouse, and human models by tobacco and other environmental exposures